30122
L-Cysteine hydrochloride monohydrate
tested according to Ph. Eur.
Synonym(s):
Cysteini hydrochloridum monohydricum
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About This Item
Linear Formula:
HSCH2CH(NH2)COOH · HCl · H2O
CAS Number:
Molecular Weight:
175.63
UNSPSC Code:
12352209
PubChem Substance ID:
eCl@ss:
32160406
EC Number:
200-157-7
Beilstein/REAXYS Number:
5158059
MDL number:
Quality Level
agency
USP/NF, tested according to Ph. Eur.
assay
98.5-101.0%
form
solid
mp
176 °C
solubility
H2O: soluble 100 g/L at 25 °C
application(s)
pharmaceutical (small molecule)
SMILES string
O.Cl.N[C@@H](CS)C(O)=O
InChI
1S/C3H7NO2S.ClH.H2O/c4-2(1-7)3(5)6;;/h2,7H,1,4H2,(H,5,6);1H;1H2/t2-;;/m0../s1
InChI key
QIJRTFXNRTXDIP-JIZZDEOASA-N
Application
L-Cysteine hydrochloride monohydrate was used in the synthesis of polycarbophil-cysteine conjugate.
Biochem/physiol Actions
Cysteine is a non-essential, thiol-containing amino acid that has protective effects in normal tissues. It is converted to glutathione that decreases the effects of chemotherapeutic and radiation agents. This might restrict the effect of therapeutic agents used for cancer treatment.
NMDA glutamatergic receptor antagonist.
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Storage Class
11 - Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
dust mask type N95 (US), Eyeshields, Gloves
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J C Roberts
Amino acids, 8(2), 113-124 (1995-06-01)
The thiol-containing amino acid, cysteine, and its analogs are useful for a variety of protective applications, including protecting normal tissues against the unwanted side effects of cancer chemotherapeutic agents and radiation treatment. The protection can result from both direct action
M K Marschütz et al.
Pharmaceutical research, 17(12), 1468-1474 (2001-04-17)
To develop an oral controlled release system for insulin. The polymer-inhibitor conjugates carboxymethylcellulose (CMC)-Bowman-Birk inhibitor and CMC-elastatinal were homogenized with polycarbophil-cysteine conjugate, insulin, and mannitol, compressed to 2 mg microtablets and enteric coated with a polymethacrylate. The protective effect of
Matthew Oser et al.
Journal of cell science, 123(Pt 21), 3662-3673 (2010-10-26)
Invadopodia are matrix-degrading membrane protrusions in invasive carcinoma cells enriched in proteins that regulate actin polymerization. The on-off regulatory switch that initiates actin polymerization in invadopodia requires phosphorylation of tyrosine residues 421, 466, and 482 on cortactin. However, it is