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Merck

203149

Copper(II) chloride

99.999% trace metals basis

동의어(들):

Cupric chloride

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제품정보 (DICE 배송 시 비용 별도)

Linear Formula:
CuCl2
CAS 번호:
7447-39-4
Molecular Weight:
134.45
PubChem Substance ID:
24852150
eCl@ss:
38150111
UNSPSC Code:
12352302
NACRES:
NA.23
EC Number:
231-210-2
MDL number:
MFCD00010972
Assay:
99.999% trace metals basis
Form:
powder

주요 문서

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InChI key

ORTQZVOHEJQUHG-UHFFFAOYSA-L

InChI

1S/2ClH.Cu/h2*1H;/q;;+2/p-2

SMILES string

Cl[Cu]Cl

assay

99.999% trace metals basis

form

powder

reaction suitability

core: copper, reagent type: catalyst

impurities

≤15.0 ppm Trace Metal Analysis

mp

620 °C (lit.)

density

3.386 g/mL at 25 °C (lit.)

Quality Level

100

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관련 카테고리

General description

Copper chloride is a white solid. It is insoluble in water. Copper(II) chloride may be prepared by dissolving excess of copper (II) oxide or copper(II) carbonate in dilute hydrochloric acid.

Application

Noble-Metal Nanostructures with Controlled Morphologies
Adding CuCl2 to AgNO3 with EG1mYAbe is used to produce uniform Ag nanowires. Copper chloride may be used as a catalyst in organic chlorination reactions. Used with palladium in a catalytic synthesis of 3-haloindoles via an annulation process.

signalword

Danger

Hazard Classifications

Acute Tox. 4 Dermal - Acute Tox. 4 Oral - Aquatic Acute 1 - Aquatic Chronic 2 - Eye Dam. 1 - Skin Irrit. 2

저장 등급

8A - Combustible corrosive hazardous materials

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

dust mask type N95 (US), Eyeshields, Gloves

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시험 성적서(COA)

Lot/Batch Number
MKBZ8739V
MKBZ0587V
MKBS7226V
MKBR3690V
MKBM6521V

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문서 라이브러리에서 최근에 구매한 제품에 대한 문서를 찾아보세요.

문서 라이브러리 방문

Noble-Metal Nanostructures with Controlled Morphologies
Jiang M, et al.
Material Matters, 4(1) null
Handbook of Copper Compounds and Applications null
Chemistry (Weinheim An Der Bergstrasse, Germany) null
Synthesis, 1841-1841 (2007)
Pieter Spincemaille et al.
Toxicology and applied pharmacology, 280(2), 345-351 (2014-08-20)
Wilson disease (WD) is caused by accumulation of excess copper (Cu) due to a mutation in the gene encoding the liver Cu transporter ATP7B, and is characterized by acute liver failure or cirrhosis and neuronal cell death. We investigated the
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