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Merck

S9663

Sodium arsenate dibasic heptahydrate

ACS reagent, ≥98%

동의어(들):

Disodium hydrogen arsenate heptahydrate, di-Sodium hydrogen arsenate heptahydrate

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제품정보 (DICE 배송 시 비용 별도)

Linear Formula:
Na2HAsO4 · 7H2O
CAS 번호:
Molecular Weight:
312.01
NACRES:
NB.24
PubChem Substance ID:
UNSPSC Code:
12352302
MDL number:
Assay:
≥98%, 98.0-102.0%
Grade:
ACS reagent
Form:
powder
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grade

ACS reagent

Quality Level

assay

≥98%, 98.0-102.0%

form

powder

impurities

≤0.005% Insoluble matter

pH

8.5-9 (25 °C, 50 g/L)

anion traces

arsenite (As2O3): ≤0.01%, chloride (Cl-): ≤0.001%, nitrate (NO3-): ≤0.005%, sulfate (SO42-): ≤0.01%

cation traces

Fe: ≤0.001%, heavy metals (as Pb): ≤0.002%

SMILES string

O.O.O.O.O.O.O.[Na+].[Na+].O[As]([O-])([O-])=O

InChI

1S/AsH3O4.2Na.7H2O/c2-1(3,4)5;;;;;;;;;/h(H3,2,3,4,5);;;7*1H2/q;2*+1;;;;;;;/p-2

InChI key

KOLXPEJIBITWIQ-UHFFFAOYSA-L

Application

Sodium arsenate dibasic heptahydrate can be used as an internal standard for the quantification of arsenic species using various spectroscopic methods.


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Danger

Hazard Classifications

Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A

저장 등급

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable



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시험 성적서(COA)

Lot/Batch Number

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문서 라이브러리 방문



William Brattin et al.
Journal of toxicology and environmental health. Part A, 76(7), 449-457 (2013-04-25)
This study describes a method for measuring the relative oral bioavailability (RBA) of arsenic (As) in soil and other soil-like media using young swine as the animal model. Groups of animals are exposed to site soil or sodium arsenate orally
Jaydip Biswas et al.
Asian Pacific journal of cancer prevention : APJCP, 11(1), 239-247 (2010-07-03)
Inorganic arsenic (As) is considered as a human carcinogen because it is associated with cancers of skin, lung, liver and bladder in exposed population. Consumption of As contaminated ground water for long term causes oxidative stress. Generation of reactive oxygen
Samantha L Goggin et al.
Neurotoxicology, 33(5), 1338-1345 (2012-09-11)
Over the past two decades, key advancements have been made in understanding the complex pathology that occurs following not only high levels of arsenic exposure (>1 ppm) but also levels previously considered to be low (<100 ppb). Past studies have